Suchergebnisse - "Wu, Xin"

PubPharm (20)

1

Impact of endometrial claudin-3 deletion on murine implantation, decidualization, and embryo development†

enthalten in: Biology of reproduction | 2022

von Grund, S. | Wu, X. | Müller, D. | +2

2

Transgene-mediated rescue of spermatogenesis in Cldn11-null mice

enthalten in: Biology of reproduction | 2012

von Wu, X. | Peppi, M. | Vengalil, M. | +4

3

Neuromedin B and its receptor influence the activity of myometrial primary cells in vitro through regulation of Il6 expression via the Rela/p65 pathway in mice

enthalten in: Biology of reproduction | 2012

von Zhang, W. | Fei, K. | Wu, M. | +2

4

Transgene-Mediated Rescue of Spermatogenesis in Cldn11-Null Mice

enthalten in: Biology of reproduction | 2012

von Wu, X. | Peppi, M. | Vengalil, M. | +4

5

Neuromedin B and Its Receptor Influence the Activity of Myometrial Primary Cells In Vitro Through Regulation of Il6 Expression via the Rela/p65 Pathway in Mice

enthalten in: Biology of reproduction | 2012

von Zhang, W. | Fei, K. | Wu, M. | +2

6

Transgene-Mediated Rescue of Spermatogenesis in Cldn11-Null Mice

enthalten in: Biology of reproduction | 2012

von Wu, X. | Peppi, M. | Vengalil, M. | +4

7

Neuromedin B and Its Receptor Influence the Activity of Myometrial Primary Cells In Vitro Through Regulation of Il6 Expression via the Rela/p65 Pathway in Mice

enthalten in: Biology of reproduction | 2012

von Zhang, W. | Fei, K. | Wu, M. | +2

8

Spermatogonial stem cell self-renewal requires ETV5-mediated downstream activation of Brachyury in mice

enthalten in: Biology of reproduction | 2011

von Wu, X. | Goodyear, S. | Tobias, J. | +2

9

Spermatogonial Stem Cell Self-Renewal Requires ETV5-Mediated Downstream Activation of Brachyury in Mice

enthalten in: Biology of reproduction | 2011

von Wu, X. | Goodyear, S. | Tobias, J. | +2

10

Spermatogonial Stem Cell Self-Renewal Requires ETV5-Mediated Downstream Activation of Brachyury in Mice

enthalten in: Biology of reproduction | 2011

von Wu, X. | Goodyear, S. | Tobias, J. | +2