Suchergebnisse - "Shu, Hong"

PubPharm (32)

1

SNX8 modulates the innate immune response to RNA viruses by regulating the aggregation of VISA

enthalten in: Cellular & molecular immunology | 2020

von Guo, W. | Wei, J. | Zhong, X. | +6

ErratumIn: Cell Mol Immunol. 2021 Jun;18(6):1613-1614. - PMID 33958747

2

Deciphering the pathways to antiviral innate immunity and inflammation

enthalten in: Advances in immunology | 2020

von Yang, Q. | Shu, H.

3

Knockout of Canopy 2 activates p16INK4a pathway to impair cardiac repair

enthalten in: Journal of molecular and cellular cardiology | 2019

von Yin, W. | Guo, J. | Zhang, C. | +7

4

USP19 Inhibits TNF-α- and IL-1β-Triggered NF-κB Activation by Deubiquitinating TAK1

enthalten in: Journal of immunology (Baltimore, Md. : 1950) | 2019

von Lei, C. | Wu, X. | Zhong, X. | +3

5

RACK1 attenuates RLR antiviral signaling by targeting VISA-TRAF complexes

enthalten in: Biochemical and biophysical research communications | 2019

von Xie, T. | Chen, T. | Li, C. | +6

ErratumIn: Biochem Biophys Res Commun. 2020 Dec 17;533(4):1529-1530. - PMID 33243463

6

TARBP2 negatively regulates IFN-β production and innate antiviral response by targeting MAVS

enthalten in: Molecular immunology | 2018

von Ling, T. | Li, S. | Weng, G. | +6

7

iRhom2 is essential for innate immunity to RNA virus by antagonizing ER- and mitochondria-associated degradation of VISA

enthalten in: PLoS pathogens | 2017

von Luo, W. | Li, S. | Li, C. | +7

8

PKACs attenuate innate antiviral response by phosphorylating VISA and priming it for MARCH5-mediated degradation

enthalten in: PLoS pathogens | 2017

von Yan, B. | Zhou, L. | Hu, M. | +5

9

TRIM32-TAX1BP1-dependent selective autophagic degradation of TRIF negatively regulates TLR3/4-mediated innate immune responses

enthalten in: PLoS pathogens | 2017

von Yang, Q. | Liu, T. | Lin, H. | +7

10

Expression of DLG1 and DLG5 in the Intestinal Epithelium of Patients with CD

enthalten in: Inflammatory bowel diseases | 2016

von Cui, H. | Shu-Hong, Y. | Qiao, M.