Suchergebnisse - "Russo, Rossella"

PubPharm (8)

1

CCR5 knockout prevents neuronal injury and behavioral impairment induced in a transgenic mouse model by a CXCR4-using HIV-1 glycoprotein 120

enthalten in: Journal of immunology (Baltimore, Md. : 1950) | 2014

von Maung, R. | Hoefer, M. | Sanchez, A. | +11

2

Isobaric tagging-based quantification by mass spectrometry of differentially regulated proteins in synaptosomes of HIV/gp120 transgenic mice : implications for HIV-associated neurodegeneration

enthalten in: Experimental neurology | 2012

von Banerjee, S. | Liao, L. | Russo, R. | +10

3

Genetic knockouts suggest a critical role for HIV co-receptors in models of HIV gp120-induced brain injury

enthalten in: Journal of neuroimmune pharmacology : the official journal of the Society on NeuroImmune Pharmacology | 2012

von Maung, R. | Medders, K. | Sejbuk, N. | +3

4

Erythropoietin plus insulin-like growth factor-I protects against neuronal damage in a murine model of human immunodeficiency virus-associated neurocognitive disorders

enthalten in: Annals of neurology | 2010

von Kang, Y. | Digicaylioglu, M. | Russo, R. | +5

5

HIV/gp120 decreases adult neural progenitor cell proliferation via checkpoint kinase-mediated cell-cycle withdrawal and G1 arrest

enthalten in: Cell stem cell | 2007

von Okamoto, S. | Kang, Y. | Brechtel, C. | +7

6

Evidence implicating matrix metalloproteinases in the mechanism underlying accumulation of IL-1beta and neuronal apoptosis in the neocortex of HIV/gp120-exposed rats

enthalten in: International review of neurobiology | 2007

von Russo, R. | Siviglia, E. | Gliozzi, M. | +5

7

17beta-estradiol protects SH-SY5Y Cells against HIV-1 gp120-induced cell death : evidence for a role of estrogen receptors

enthalten in: Neurotoxicology | 2005

von Russo, R. | Navarra, M. | Maiuolo, J. | +3

8

Evidence for a role of protein tyrosine kinases in cell death induced by gp120 in CHP100 neuroblastoma cells

enthalten in: Toxicology letters | 2003

von Russo, R. | Navarra, M. | Rotiroti, D. | +1

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