Suchergebnisse - "Qi, Jiansong"

PubPharm (29)

1

MCOLN1/TRPML1 in the lysosome : a promising target for autophagy modulation in diverse diseases

enthalten in: Autophagy | 2024

von Qi, J. | Li, Q. | Xin, T. | +9

2

NCK1 Modulates Neuronal Actin Dynamics and Promotes Dendritic Spine, Synapse, and Memory Formation

enthalten in: The Journal of neuroscience : the official journal of the Society for Neuroscience | 2023

von Diab, A. | Wigerius, M. | Quinn, D. | +7

3

Targeting MCOLN1/TRPML1 channels to protect against ischemia-reperfusion injury by restoring the inhibited autophagic flux in cardiomyocytes

enthalten in: Autophagy | 2022

von Sui, Z. | Wang, M. | Xing, Y. | +2

4

TRPML1-induced autophagy inhibition triggers mitochondrial mediated apoptosis

enthalten in: Cancer letters | 2022

von Liu, Y. | Wang, X. | Zhu, W. | +6

5

Autophagy inhibition mediated by MCOLN1/TRPML1 suppresses cancer metastasis via regulating a ROS-driven TP53/p53 pathway

enthalten in: Autophagy | 2022

von Xing, Y. | Wei, X. | Liu, Y. | +14

6

Blunting TRPML1 channels protects myocardial ischemia/reperfusion injury by restoring impaired cardiomyocyte autophagy

enthalten in: Basic research in cardiology | 2022

von Xing, Y. | Sui, Z. | Liu, Y. | +14

7

Blunting TRPML1 channels protects myocardial ischemia/reperfusion injury by restoring impaired cardiomyocyte autophagy

enthalten in: Basic research in cardiology | 2022

von Xing, Y. | Sui, Z. | Liu, Y. | +14

8

Blunting TRPML1 channels protects myocardial ischemia/reperfusion injury by restoring impaired cardiomyocyte autophagy

enthalten in: Basic research in cardiology | 2022

von Xing, Y. | Sui, Z. | Liu, Y. | +14

9

Blunting TRPML1 channels protects myocardial ischemia/reperfusion injury by restoring impaired cardiomyocyte autophagy

enthalten in: Basic research in cardiology | 2022

von Xing, Y. | Sui, Z. | Liu, Y. | +14

10

Stimulating TRPM7 suppresses cancer cell proliferation and metastasis by inhibiting autophagy

enthalten in: Cancer letters | 2022

von Xing, Y. | Wei, X. | Wang, M. | +17