Suchergebnisse - "Mescia, Federica"

PubPharm (52)

1

Factor V is an immune inhibitor that is expressed at increased levels in leukocytes of patients with severe Covid-19

enthalten in: bioRxiv.org | 2024

von Wang, J. | Kotagiri, P. | Lyons, P. | +27

2

The cellular immune response to COVID-19 deciphered by single cell multi-omics across three UK centres

enthalten in: bioRxiv.org | 2024

von Stephenson, E. | Reynolds, G. | Botting, R. | +65

3

A patient-centric characterization of systemic recovery from SARS-CoV-2 infection

enthalten in: bioRxiv.org | 2024

von Ruffieux, H. | Hanson, A. | Lodge, S. | +16

4

Proteomic analysis of circulating immune cells identifies novel cellular phenotypes associated with COVID-19 severity

enthalten in: bioRxiv.org | 2024

von Potts, M. | Fletcher-Etherington, A. | Nightingale, K. | +14

5

Iron dysregulation and inflammatory stress erythropoiesis associates with long-term outcome of COVID-19

enthalten in: Nature immunology | 2024

von Hanson, A. | Mulè, M. | Ruffieux, H. | +14

6

Sustained post-rituximab B-cell depletion is common in ANCA-associated vasculitis and is affected by sex and renal function

enthalten in: Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association | 2024

von Mescia, F. | Salviani, C. | Tonoli, M. | +13

7

A critical view on autoantibodies in lupus nephritis : Concrete knowledge based on evidence

enthalten in: Autoimmunity reviews | 2024

von Bruschi, M. | Angeletti, A. | Prunotto, M. | +46

8

Clinical Significance of the Cystic Phenotype in Alport Syndrome

enthalten in: American journal of kidney diseases : the official journal of the National Kidney Foundation | 2024

von Zeni, L. | Mescia, F. | Toso, D. | +9

9

Proinflammatory cytokines driving cardiotoxicity in COVID-19

enthalten in: Cardiovascular research | 2024

von Colzani, M. | Bargehr, J. | Mescia, F. | +8

CommentIn: Cardiovasc Res. 2024 Jan 25;:. - PMID 38270957

10

Monoallelic pathogenic IFT140 variants are a common cause of autosomal dominant polycystic kidney disease-spectrum phenotype

enthalten in: Clinical kidney journal | 2024

von Dordoni, C. | Zeni, L. | Toso, D. | +8

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