Suchergebnisse - "Lugenbiel, Patrick"

PubPharm (8)

1

Cardiac K2P13.1 (THIK-1) two-pore-domain K+ channels : Pharmacological regulation and remodeling in atrial fibrillation

enthalten in: Progress in biophysics and molecular biology | 2019

von Staudacher, I. | Seehausen, S. | Illg, C. | +4

2

Cardiac K2P13.1 (THIK-1) two-pore-domain K+ channels : pharmacological regulation and remodeling in atrial fibrillation

enthalten in: Progress in biophysics & molecular biology | 6 July 2018

von Staudacher, I. | Seehausen, S. | Illg, C. | +4

3

TREK-1 (K2P2.1) K+ channels are suppressed in patients with atrial fibrillation and heart failure and provide therapeutic targets for rhythm control

enthalten in: Basic research in cardiology | 2017

von Lugenbiel, P. | Wenz, F. | Syren, P. | +14

4

TREK-1 (K2P2.1) K+ channels are suppressed in patients with atrial fibrillation and heart failure and provide therapeutic targets for rhythm control

enthalten in: Basic research in cardiology | 2017

von Lugenbiel, P. | Wenz, F. | Syren, P. | +14

5

TREK-1 ($ K_{2P} $2.1) $ K^{+} $ channels are suppressed in patients with atrial fibrillation and heart failure and provide therapeutic targets for rhythm control

enthalten in: Basic research in cardiology | 2016

von Lugenbiel, P. | Wenz, F. | Syren, P. | +14

6

TREK-1 ($ K_{2P} $2.1) $ K^{+} $ channels are suppressed in patients with atrial fibrillation and heart failure and provide therapeutic targets for rhythm control

enthalten in: Basic research in cardiology | 2016

von Lugenbiel, P. | Wenz, F. | Syren, P. | +14

7

Cloning, functional characterization, and remodeling of K2P3.1 (TASK-1) potassium channels in a porcine model of atrial fibrillation and heart failure

enthalten in: Heart rhythm | 2014

von Schmidt, C. | Wiedmann, F. | Langer, C. | +7

CommentIn: Heart Rhythm. 2014 Oct;11(10):1806-7. - PMID 25041966

8

Cardiac expression and atrial fibrillation-associated remodeling of K₂p2.1 (TREK-1) K⁺ channels in a porcine model

enthalten in: Life sciences | 2014

von Schmidt, C. | Wiedmann, F. | Tristram, F. | +6

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