Suchergebnisse - "Kyttaris, Vasileios C"

PubPharm (46)

1

The role of IL-23/IL-17 axis in lupus nephritis

enthalten in: Journal of immunology (Baltimore, Md. : 1950) | 2009

von Zhang, Z. | Kyttaris, V. | Tsokos, G.

2

The Role of IL-23-IL-17 Axis in Lupus Nephritis

enthalten in: The journal of immunology | 2009

von Zhang, Z. | Kyttaris, V. | Tsokos, G.

3

Expanded double negative T cells in patients with systemic lupus erythematosus produce IL-17 and infiltrate the kidneys

enthalten in: Journal of immunology (Baltimore, Md. : 1950) | 2008

von Crispín, J. | Oukka, M. | Bayliss, G. | +6

4

PP2A dephosphorylates Elf-1 and determines the expression of CD3zeta and FcRgamma in human systemic lupus erythematosus T cells

enthalten in: Journal of immunology (Baltimore, Md. : 1950) | 2008

von Juang, Y. | Wang, Y. | Jiang, G. | +6

5

T cells and in situ cryoglobulin deposition in the pathogenesis of lupus nephritis

enthalten in: Clinical immunology (Orlando, Fla.) | 2008

von Cohen, R. | Bayliss, G. | Crispin, J. | +7

6

The RNA-stabilizing protein HuR regulates the expression of zeta chain of the human T cell receptor-associated CD3 complex

enthalten in: The Journal of biological chemistry | 2008

von Moulton, V. | Kyttaris, V. | Juang, Y. | +2

7

T cells and in situ cryoglobulin deposition in the pathogenesis of lupus nephritis

enthalten in: Clinical immunology | 2008

von Cohen, R. | Bayliss, G. | Crispin, J. | +7

8

How signaling and gene transcription aberrations dictate the systemic lupus erythematosus T cell phenotype

enthalten in: Trends in immunology | 2008

von Crispín, J. | Kyttaris, V. | Juang, Y. | +1

9

How signaling and gene transcription aberrations dictate the systemic lupus erythematosus T cell phenotype

enthalten in: Trends in immunology | 2008

von Crispín, J. | Kyttaris, V. | Juang, Y. | +1

10

How signaling and gene transcription aberrations dictate the systemic lupus erythematosus T cell phenotype

enthalten in: Trends in immunology | 2008

von Crispín, J. | Kyttaris, V. | Juang, Y. | +1

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