Hepatic Vasculopathy and Regenerative Responses of the Liver in Fatal Cases of COVID-19
Copyright © 2021 The Authors. Published by Elsevier Inc. All rights reserved..
Severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2) infects the nasopharynx and lungs and causes coronavirus disease-2019 (COVID-19). It may impact the heart, brain, kidney, and liver.1 Although functional impairment of the liver has been correlated with worse clinical outcomes, little is known about the pathophysiology of hepatic injury and repair in COVID-19.2,3 Histologic evaluation has been limited to small numbers of COVID-19 cases with no control subjects2,4 and demonstrated largely heterogeneous patterns of pathology.2,3.
Errataetall: |
CommentIn: Clin Gastroenterol Hepatol. 2021 Jul;19(7):1508-1509. - PMID 33581360 |
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Medienart: |
E-Artikel |
Erscheinungsjahr: |
2021 |
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Erschienen: |
2021 |
Enthalten in: |
Zur Gesamtaufnahme - volume:19 |
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Enthalten in: |
Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association - 19(2021), 8 vom: 01. Aug., Seite 1726-1729.e3 |
Sprache: |
Englisch |
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Beteiligte Personen: |
Kaltschmidt, Barbara [VerfasserIn] |
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Date Completed 18.08.2021 Date Revised 03.04.2024 published: Print-Electronic CommentIn: Clin Gastroenterol Hepatol. 2021 Jul;19(7):1508-1509. - PMID 33581360 Citation Status MEDLINE |
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doi: |
10.1016/j.cgh.2021.01.044 |
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funding: |
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Förderinstitution / Projekttitel: |
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PPN (Katalog-ID): |
NLM320782638 |
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520 | |a Severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2) infects the nasopharynx and lungs and causes coronavirus disease-2019 (COVID-19). It may impact the heart, brain, kidney, and liver.1 Although functional impairment of the liver has been correlated with worse clinical outcomes, little is known about the pathophysiology of hepatic injury and repair in COVID-19.2,3 Histologic evaluation has been limited to small numbers of COVID-19 cases with no control subjects2,4 and demonstrated largely heterogeneous patterns of pathology.2,3 | ||
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700 | 1 | |a Fitzek, Antonia D E |e verfasserin |4 aut | |
700 | 1 | |a Schaedler, Julia |e verfasserin |4 aut | |
700 | 1 | |a Förster, Christine |e verfasserin |4 aut | |
700 | 1 | |a Kaltschmidt, Christian |e verfasserin |4 aut | |
700 | 1 | |a Hansen, Torsten |e verfasserin |4 aut | |
700 | 1 | |a Steinfurth, Fabian |e verfasserin |4 aut | |
700 | 1 | |a Windmöller, Beatrice A |e verfasserin |4 aut | |
700 | 1 | |a Pilger, Christian |e verfasserin |4 aut | |
700 | 1 | |a Kong, Cihang |e verfasserin |4 aut | |
700 | 1 | |a Singh, Kashika |e verfasserin |4 aut | |
700 | 1 | |a Nierhaus, Axel |e verfasserin |4 aut | |
700 | 1 | |a Wichmann, Dominic |e verfasserin |4 aut | |
700 | 1 | |a Sperhake, Jan |e verfasserin |4 aut | |
700 | 1 | |a Püschel, Klaus |e verfasserin |4 aut | |
700 | 1 | |a Huser, Thomas |e verfasserin |4 aut | |
700 | 1 | |a Krüger, Martin |e verfasserin |4 aut | |
700 | 1 | |a Robson, Simon C |e verfasserin |4 aut | |
700 | 1 | |a Wilkens, Ludwig |e verfasserin |4 aut | |
700 | 1 | |a Schulte Am Esch, Jan |e verfasserin |4 aut | |
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