Neurologic compromise in pediatric COVID-19 infection
Coronavirus disease has extended its involvement beyond the respiratory system, with increasing reports of involving different systems, such as Nervous System. The neuroinvasive potential of this pathogen would be explained by its neurotropism given the presence of ACE2 receptors in the brain and spinal cord, in addition to the important systemic inflammatory involvement. The neu rological involvement due to infection is divided between the central nervous system, highlighting non-specific and mild symptoms such as dizziness and headache, as well as severe symptoms with encephalitis and cerebrovascular pathology, and the peripheral nervous system, which mainly pre sents anosmia, ageusia, and myositis. Clinical symptomatology in pediatric patients seems to be less than in adults, but there is a growing report in the literature regarding these findings. There fore, it is very important to have an adequate registry and anamnesis that allow early identification of neurological involvement.
Errataetall: |
CommentIn: Andes Pediatr. 2021 Feb;92(1):151-152. - PMID 34106196 |
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Medienart: |
E-Artikel |
Erscheinungsjahr: |
2020 |
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Erschienen: |
2020 |
Enthalten in: |
Zur Gesamtaufnahme - volume:91 |
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Enthalten in: |
Revista chilena de pediatria - 91(2020), 4 vom: 05. Aug., Seite 614-619 |
Sprache: |
Spanisch |
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Weiterer Titel: |
Compromiso neurológico en infección por COVID-19 en pacientes pediátricos |
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Beteiligte Personen: |
Quiroz S, Vicente [VerfasserIn] |
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Links: |
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Themen: |
ACE2 protein, human |
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Anmerkungen: |
Date Completed 11.01.2021 Date Revised 11.06.2021 published: Print-Electronic CommentIn: Andes Pediatr. 2021 Feb;92(1):151-152. - PMID 34106196 Citation Status MEDLINE |
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doi: |
10.32641/rchped.vi91i4.2526 |
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funding: |
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Förderinstitution / Projekttitel: |
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PPN (Katalog-ID): |
NLM319636232 |
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520 | |a Coronavirus disease has extended its involvement beyond the respiratory system, with increasing reports of involving different systems, such as Nervous System. The neuroinvasive potential of this pathogen would be explained by its neurotropism given the presence of ACE2 receptors in the brain and spinal cord, in addition to the important systemic inflammatory involvement. The neu rological involvement due to infection is divided between the central nervous system, highlighting non-specific and mild symptoms such as dizziness and headache, as well as severe symptoms with encephalitis and cerebrovascular pathology, and the peripheral nervous system, which mainly pre sents anosmia, ageusia, and myositis. Clinical symptomatology in pediatric patients seems to be less than in adults, but there is a growing report in the literature regarding these findings. There fore, it is very important to have an adequate registry and anamnesis that allow early identification of neurological involvement | ||
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