Suchergebnisse - (Person:"Shen, Han")

PubPharm (12)

1

IinQ attenuates systemic inflammatory responses via selectively impairing the Myddosome complex formation upon TLR4 ligation

enthalten in: Biochemical pharmacology | 2016

von Kang, K. | Won, M. | Yuk, J. | +8

2

IinQ attenuates systemic inflammatory responses via selectively impairing the Myddosome complex formation upon TLR4 ligation

enthalten in: Biochemical pharmacology | 2016

von Kang, K. | Won, M. | Yook, J. | +8

3

IinQ attenuates systemic inflammatory responses via selectively impairing the Myddosome complex formation upon TLR4 ligation

enthalten in: Biochemical pharmacology | 2016

von Kang, K. | Won, M. | Yuk, J. | +8

4

Inhibition of the JAK-STAT3 pathway by andrographolide enhances chemosensitivity of cancer cells to doxorubicin

enthalten in: Biochemical pharmacology | 2010

von Zhou, J. | Ong, C. | Hur, G. | +1

5

Endoplasmic reticulum calcium release potentiates the ER stress and cell death caused by an oxidative stress in MCF-7 cells

enthalten in: Biochemical pharmacology | 2010

von Zhou, J. | Ong, C. | Hur, G. | +1

6

Critical role of Bid and Bax in indirubin-3'-monoxime-induced apoptosis in human cancer cells

enthalten in: Biochemical pharmacology | 2008

von Shi, J. | Shen, H.

7

Critical role of Bid and Bax in indirubin-3′-monoxime-induced apoptosis in human cancer cells

enthalten in: Biochemical pharmacology | 2008

von Shi, J. | Shen, H.

8

Critical role of pro-apoptotic Bcl-2 family members in andrographolide-induced apoptosis in human cancer cells

enthalten in: Biochemical pharmacology | 2006

von Zhou, J. | Zhang, S. | Ong, C. | +1

9

Critical role of pro-apoptotic Bcl-2 family members in andrographolide-induced apoptosis in human cancer cells

enthalten in: Biochemical pharmacology | 2006

von Zhou, J. | Zhang, S. | Choon-Nam, O. | +1

10

Inhibitory effect of emodin on tumor invasion through suppression of activator protein-1 and nuclear factor-kappaB

enthalten in: Biochemical pharmacology | 2004

von Huang, Q. | Shen, H. | Ong, C.